Juniper Publishers Journal of Dairy and Veterinary Sciences

JUNIPER PUBLISHERS - OPEN ACCESS JOURNAL OF DAIRY & VETERINARY SCIENCES Abstract This review article provides a brief description of the nutritional bases to control gastrointestinal (GI) parasites in livestock and maintained/ improved their production potential by nutritional intervention. High prevalence of GI parasites, resistance against synthetic drugs, high prices, and scarcity of good quality feedstuffs may pose the disease problem with great economic losses worldwide. The GI parasites infestation may impose progressive decline in feed intake and impaired efficient nutrient utilization, loss in body weight, diarrhea, malnutrition, sometimes tissue damage which leads to blood loss (anemia), various nutrient deficiency and sometimes death may occur Due to infection stress, nutrient intake often compromised and available nutrients were diverted away from production purposes to a high survival priority i.e. for tissue wear and tear/ repair, synthesis of immunoglo

JUNIPER PUBLISHERS - OPEN ACCESS JOURNAL OF DAIRY & VETERINARY SCIENCES   Introduction 2,3,7,8-Tetrachlorodibenzo-p-dioxin (TCDD), commonly known as dioxin, is a widespread, persistent, and toxic environmental pollutant, which can suppress immune response thereby leading to an increased susceptibility to infectious agents, including Bovine herpesvirus-1 (BoHV-1). BoHV-1 is an important cattle pathogen that can cause infectious bovine rhinotracheitis (IBR), infectious pustular vulvovaginitis (IPV), conjunctivitis and abortions. Following infection in bovine cells (MDBK), TCDD significantly enhances BoHV-1 replication, through an anticipation of BoHV-1-induced apoptosis, by interfering with iron homeostasis and stimulating the expression of bICP0, the major regulatory protein of BoHV-1. As consequence, TCDD may be an additional threat for BoHV-1 infection in animals. Dioxin is a persistent environmental contaminant which may induce toxicity, both in humans and in